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Correspondence
3 Results
- Correspondence
Murine typhus presenting as pseudotumor cerebri
Canadian Journal of OphthalmologyVol. 56Issue 4e131–e133Published online: January 30, 2021- Sugi Panneerselvam
- Nita Bhat
- Shruthi Harish Bindiganavile
- Andrew G. Lee
Cited in Scopus: 0Murine typhus is an acute infection transmitted by rodent or cat fleas carrying gram-negative, obligate intracellular bacteria, Rickettsia typhi. This vector for transmission is most often carried on rodents, but opossums are thought to be a reservoir in suburban settings.1 Associated with overcrowding, pollution, and poor hygiene, murine typhus has been documented worldwide, but the majority of cases in the United States are in Texas and California.2 - Correspondence
Convergence spasm with horizontal nystagmus in anti-GAD65 antibody syndrome
Canadian Journal of OphthalmologyVol. 56Issue 1e20–e21Published online: July 20, 2020- Beena M. Shah
- Nita Bhat
- Shruthi Harish Bindiganavile
- Andrew G. Lee
Cited in Scopus: 1Anti-glutamic acid decarboxylase (anti-GAD) autoantibody syndrome produces symptoms and signs related to loss of the inhibitory neurotransmitter gamma aminobutyric acid. Patients with GAD antibody may present with hyperexcitability disorders, including stiff-person syndrome, limbic encephalitis, and nystagmus. There has been only one previous report of convergence spasm in a patient with GAD antibody. Here we present a case of convergence spasm and, to our knowledge, the first case in the English-language ophthalmic literature to have concomitant horizontal nystagmus, in a patient with anti-GAD65 antibody syndrome. - Correspondence
Dysgeusia and amaurosis fugax: a unique presentation in spontaneous internal carotid artery dissection
Canadian Journal of OphthalmologyVol. 55Issue 6e205–e207Published online: June 22, 2020- Preston Baker
- Shruthi Harish Bindiganavile
- Ross Taylor
- Nita Bhat
- Andrew G. Lee
Cited in Scopus: 0Spontaneous internal carotid artery (ICA) dissection is a significant cause of ischemic stroke in young adults1. The presenting symptoms and signs of ICA dissection (ICAD) are variable and can be due to direct local involvement of neural structures (e.g., oculosympathetic plexus causing the ipsilateral Horner syndrome) or indirect distal ischemia to brain or eye (e.g., transient ischemic attack or stroke) from thromboembolic disease 23. Transient monocular visual loss (TMVL) and the Horner syndrome are well-known presentations of ICAD and may occur in up to 50% of cases 4.